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The newly developed dual agonists, which activate both GLP1R and glucose-dependent insulinotropic peptide (GIP) receptor (GIPR), appear more efficacious than GLP1RA alone in reducing HbA1C and body weight [116]
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26 It promotes satiety and reduces food intake by activating GLP-1 receptors (GLP-1R) in the arcuate nucleus of the hypothalamus, stimulating neurons that release satiety signals such as proopiomelanocortin
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